Telomere length, c-myc and mad-1 expression could represent prognosis markers of myelodysplastic syndrome

Antonella Poloni, Federica Serrani, Eleonora Berardinelli, Giulia Maurizi, Marianna Mariani, Benedetta Costantini, Silvia Trappolini, Stefania Mancini, Attilio Olivieri, Pietro Leoni

Research output: Contribution to journalArticlepeer-review

Abstract

Telomere dysfunction might generate genomic instability leading to the progression of myelodysplastic syndromes (MDS) into acute myeloid leukemia (AML). We investigated telomere length (TL), telomerase activity (TA) and hTERT, c-myc, mad1, and p53 expression in the bone marrow of patients with MDS (n= 109), AML (n= 47) and in controls (n= 24). TL was lower in MDS patients than in controls (p<0.001) and higher in L-MDS (low, intermediate-1 IPSS, p<0.01) respect H-MDS (high, intermediate-2 IPSS, p<0.01) patients. Mad-1 expression was higher in MDS patients than in controls (p<0.01), c-myc expression was highest in AML and in H-MDS patients. Our results show that the telomere dynamics might be useful for stratifying patients according to a risk scoring system.

Original languageEnglish
Pages (from-to)1538-1544
Number of pages7
JournalLeukemia Research
Volume37
Issue number11
DOIs
Publication statusPublished - Nov 2013

Keywords

  • Acute myeloid leukemia
  • HTERT
  • Myelodysplastic syndromes
  • Telomerase activity
  • Telomere length
  • Transcription factors

ASJC Scopus subject areas

  • Cancer Research
  • Hematology
  • Oncology

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