Activated DDR1 increases RS cell survival

Antonino Carbone; Annunziata Gloghini

doi:10.1182/blood-2013-11-534123

Activated DDR1 increases RS cell survival

Antonino Carbone, Annunziata Gloghini

Research output: Contribution to journal › Article › peer-review

Abstract

In this issue of Blood, Cader et al show that tumor microenvironment promotes Epstein-Barr virus (EBV)-driven lymphomagenesis in Hodgkin lymphoma by a novel pathway involving latent membrane protein 1 (LMP1) and discoidin domain receptor 1 (DDR1), which is activated by collagen(s) and contributes to the survival of Reed-Sternberg (RS) cells.

Original language	English
Pages (from-to)	4152-4154
Number of pages	3
Journal	Blood
Volume	122
Issue number	26
DOIs	https://doi.org/10.1182/blood-2013-11-534123
Publication status	Published - Dec 19 2013

ASJC Scopus subject areas

Hematology
Biochemistry
Cell Biology
Immunology

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10.1182/blood-2013-11-534123

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title = "Activated DDR1 increases RS cell survival",

abstract = "In this issue of Blood, Cader et al show that tumor microenvironment promotes Epstein-Barr virus (EBV)-driven lymphomagenesis in Hodgkin lymphoma by a novel pathway involving latent membrane protein 1 (LMP1) and discoidin domain receptor 1 (DDR1), which is activated by collagen(s) and contributes to the survival of Reed-Sternberg (RS) cells.",

author = "Antonino Carbone and Annunziata Gloghini",

year = "2013",

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doi = "10.1182/blood-2013-11-534123",

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AB - In this issue of Blood, Cader et al show that tumor microenvironment promotes Epstein-Barr virus (EBV)-driven lymphomagenesis in Hodgkin lymphoma by a novel pathway involving latent membrane protein 1 (LMP1) and discoidin domain receptor 1 (DDR1), which is activated by collagen(s) and contributes to the survival of Reed-Sternberg (RS) cells.

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Activated DDR1 increases RS cell survival

Abstract

ASJC Scopus subject areas

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